Functional TRPV6 channels are crucial for transepithelial Ca absorption

نویسندگان

  • Titia E. Woudenberg-Vrenken
  • Anke L. Lameris
  • Petra Weißgerber
  • Jenny Olausson
  • Veit Flockerzi
  • René J. M. Bindels
  • Marc Freichel
  • Joost G. J. Hoenderop
چکیده

Titia E. Woudenberg-Vrenken, Anke L. Lameris,* Petra Weißgerber,* Jenny Olausson, Veit Flockerzi, René J. M. Bindels, Marc Freichel, and Joost G. J. Hoenderop Department of Physiology, Radboud University Nijmegen Medical Centre, Nijmegen, The Netherlands; Experimentelle und Klinische Pharmakologie und Toxikologie, Universität des Saarlandes, Homburg, Germany; and Pharmakologisches Institut, Universität Heidelberg, Heidelberg, Germany

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Functional TRPV6 channels are crucial for transepithelial Ca2+ absorption.

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BACKGROUND TRPV6 ion channels are key mediators of regulated transepithelial absorption of Ca2+ within the small intestine. Trpv6-/- mice were reported to have lower bone density than wild-type littermates and significant disturbances in calcium homeostasis that suggested a role for TRPV6 in osteoblasts during bone formation and mineralization. TRPV6 and molecules related to transepithelial Ca2...

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TRPV6 is a member of the transient receptor potential superfamily of ion channels that facilitates Ca(2+) absorption in the intestines. These channels display high selectivity for Ca(2+), but in the absence of divalent cations they also conduct monovalent ions. TRPV6 channels have been shown to be inactivated by increased cytoplasmic Ca(2+) concentrations. Here we studied the mechanism of this ...

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Transient receptor potential vanilloid 6 (TRPV6) channels play an important role in intestinal Ca(2+) transport. These channels undergo Ca(2+)-induced inactivation. Here we show that Ca(2+) flowing through these channels activates phospholipase C (PLC) leading to the depletion of phosphatidylinositol 4,5-bisphosphate (PIP(2)) and formation of inositol 1,4,5-trisphosphate in TRPV6-expressing cel...

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تاریخ انتشار 2012